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658. Udager AM, McHugh JB, Betz BL, et al. Activating KRAS mutations are characteristic of oncocytic sinonasal papil loma and associated sinonasal squamous cell carcinoma: KRAS -mutated oncocytic sinonasal tumours. J Pathol . 2016;239(4):394-398. https://doi.org/10.1002/path.4750 659. Udager AM, Rolland DCM, McHugh JB, et al. High-frequency targetable EGFR mutations in sinonasal squamous cell car cinomas arising from inverted sinonasal papilloma. Cancer Res . 2015;75(13):2600-2606. https://doi.org/10.1158/0008-5472. CAN-15-0340 660. Brown NA, Plouffe KR, Yilmaz O, et al. TP53 mutations and CDKN2A mutations/deletions are highly recurrent molecular alterations in the malignant progression of sinonasal papillo mas. ModPathol . 2021;34(6):1133-1142. https://doi.org/10.1038/ s41379-020-00716-3 661. Nishikawa D, Sasaki E, Suzuki H, et al. Treatment outcome and pattern of recurrence of sinonasal squamous cell car cinoma with EGFR-mutation and human papillomavirus. J Craniomaxillofac Surg . 2021;49(6):494-500. https://doi.org/10. 1016/j.jcms.2021.02.016 662. Rooper LM, Bishop JA, Westra WH. Transcriptionally active high-risk human papillomavirus is not a common etiologic agent in the malignant transformation of inverted Schnei derian papillomas. Head and Neck Pathol . 2017;11(3):346-353. https://doi.org/10.1007/s12105-017-0779-0 663. Stepp WH, Farzal Z, Kimple AJ, et al. HPV in the malig nant transformation of sinonasal inverted papillomas: a meta-analysis. Int Forum Allergy Rhinol . 2021;11(10):1461-1471. https://doi.org/10.1002/alr.22810 664. Lewis JS, Westra WH, Thompson LDR, et al. The sinonasal tract: another potential “hot spot” for carcinomas with transcriptionally-active human papillomavirus. Head and Neck Pathol . 2014;8(3):241-249. https://doi.org/10.1007/s12105 013-0514-4 665. Tong CCL, Palmer JN. Updates in the cause of sinonasal inverted papilloma and malignant transformation to squamous cell carcinoma. Curr Opin Otolaryngol Head Neck Surg . 2021;29(1):59-64. https://doi.org/10.1097/MOO. 0000000000000692 666. Mehrad M, Stelow EB, Bishop JA, et al. Transcription ally active HPV and targetable EGFR mutations in sinonasal inverted papilloma: an association between low-risk HPV, condylomatous morphology, and can cer risk? Am J Surg Pathol . 2020;44(3):340-346. https://doi.org/10.1097/PAS.0000000000001411 667. Udager AM, McHugh JB, Goudsmit CM, et al. Human papil lomavirus (HPV) and somatic EGFR mutations are essen tial, mutually exclusive oncogenic mechanisms for inverted sinonasal papillomas and associated sinonasal squamous cell carcinomas. AnnOncol . 2018;29(2):466-471. https://doi.org/10. 1093/annonc/mdx736 668. Udager AM, McHugh JB, Elenitoba-Johnson KSJ, Brown NA. EGFR mutations in sinonasal squamous tumors: oncogenic and therapeutic implications. Oncoscience . 2015;2(11):908-909. https://doi.org/10.18632/oncoscience.268 669. Cabal VN, Menendez M, Vivanco B, et al. EGFR mutation and HPV infection in sinonasal inverted papilloma and squamous cell carcinoma. Rhinology . 2020;58(4):368-376. https://doi.org/ 10.4193/Rhin19.371